Chronic Fatigue Syndrome: Advancing Research and Clinical Education

Doctors and scientists have not yet found what causes chronic fatigue syndrome. Infections and other medical and biological events are among the many possible triggers, but with no certain cause this disease is difficult to diagnose. A systematic approach to evaluating people with persistent symptoms combined with new diagnostic criteria offers hope for more accurate and faster diagnosis.

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5 Replies to “Chronic Fatigue Syndrome: Advancing Research and Clinical Education”

  1. This is a very medical disease, not psychological. My son was straight A Stanford student 2 years ago, got ill with virus, triggered M.E. =Myalgic Encephalomyelitis, an internationally accepted disease ICD G93.3. Now he cannot talk, chew, listed or care for himself. He did not wish himself to get M.E., so Dr. Lapp and similar old timers need to stop promoting this myth esp for Severe M.E. and vast majority of others. The CDC and NIH need to use internationally accepted M.E. and stop making other vague terms that make this hard to study to find biomarkers, mechanisms, treatments, cures. NIH need much more funding to help 2 million neglected in US. Am thrilled that other organizational like Open Medicine have found private funding to make up for decades or gross neglect by NIH and CDC, and they have started Sever M.E. Biomarker study (see End-MECFS.org). Hope NIH can increase funding for M.E. as Dr. Francis Collins has promised, and fund both internal and external research, since NIH does not have all the answers, and at times, like last 3 decades, can have difficulty thinking outside their box. Good to see some movement incl Dr. Nath, but Lapp and Dr. Walitt are petrifying if they have outdated attitudes that this still psych disease, and not recognize realities like POTS, high urine output, low natural killer cells, abnormal Cytokines, low blood volumes, brain abnormalities, qEEG abnormalities (see work by Zinns), SPECT and MRI defects, many similarities and some differences vs MS (Great work in Australia!), Biome issues, viral triggers and other quantifiable physiological issues.

  2. Endurance athlete 10 years, struck down at 33y/o. Had cfs 2 and a bit years now. Horrendous experience. Thought I was dying. Infectious onset. Am slowly making good progress and consider myself a mild case (was severe in beginning) now. This illness has taken me beyond my limits on every level and still it persists. I am hopeful I can get back to work within the next year. I am using LDN.

  3. 22 year old pre-med student here. After getting mono freshman year, my acute state degenerated into CFS. It's been hell but here I am 3 years later and my condition has almost completely resolved, likely due to the resilience of my youthful immune system, a privilege many CFS sufferers do not have. I have written a 28 page thesis on the pathogenesis of CFS based on a 2 year thorough meta analysis of all available research and it is awaiting publication. Hopefully it will help to educate the clinical community and instate actual clinical parameters using epigenetic diagnostic tools so that doctors can stop approaching CFS as a psychosomatic affect.

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